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Lack of up-regulation of key antioxidative enzymes during HS indicates that ctl-2(ua90)II strain does not fully sense oxidative stress during HS, presumably as a consequence of catalase deficiency, or is unable to respond properly

Visual function limited only by tumor location and secondary complications

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The mechanism through which they are coupled is not obvious from our structures alone, though we hypothesise that the two glutamine residues previously identified as important for sodium-dependent transport 46 , Q180 and Q207, play a role in linking the Na + and carnitine sites